Tapuni

pour mon experience je peut te dire que tu farais un peu de fatigue maintenot a le trasporter mais serait tout benefice quand il serait vieux moi j'ai pas fait faire escalier a ma princesse Anthos fin a l'age de 8/9 mois et je te peut assurer que a 12 ans elle faisait ancore les escalier (doucement mais elle ete capable de le faire!) pour mon bebè male Fanfan fin a 9 mois on l'a ammenè dans le bras toutes les soir pour dormir a l'etage seulement maintenot que il a 13 mois il a droit a monter et descendre tout seul meme chose pour la voiture mon mari a preparè un truc pour le faire monter et descendre da ma voiture pour la sienne (4x4) on le prend ancore maintenot en bras pour monter et descendre

super papi Reno

j'espere que il trouve vite une maison

si peut te aider http://www.helpemup.com/flashmovie.html

idem

j'espere qui il trouve un maison pouvre loulou

generalment quand une femelle est incinte il faut une alimentation plus riche! avec de la viande etc etc

pour moi c'est ok pour la ballade le 6 june on effet par contre je trouve que l'idee de aller a Fontainebleau a la journai venerie soit mimi alors les filles vous voulez faire qua?

alè vite une maison pour ce boubou!!

ok c'est surtout sur papier que je pense il faut chercher sur internet sur les vieilles lignèè y'l a presque rien! je sais parce que pour mon Anthos dejà c'ete difficil!!! Le date base de la Berner garde peut etre utile a chacun entre nous remplisse le formulaire!!!!!! il peut nous aider pour la recherche des maladie etc etc bisou Rita

ahhahah genial alors ecoute moi j'ai fait aussi une collecte d'argent en favour the Berner garde foundatio, en octobre ici moi Fanfan e son elleveuse J'ecris avec les membres de la berner gard en particolarment avec Mme Pat Long je te donne moi le bonne adresse et n'oublie pas de inserer tes boubous mon Anthos est fichè, mon Fanfan pas ancore parce que il est pas ancore adult ici le link http://www.bernergarde.org/home/default.aspx

meri beacoup, a dire le vrai c'est là je l'avait trouvais moi aussi j'esperè d'avoir quelque reinsegnement en plus, mais sur le net y'l a rien!! merci merci bisu Rita

courage et caresse a Abby

Une amie des USA m'a domandè si gentilment quelqun a des indormations ou photos sur Arianne Riaz mere de Marquis des Cintres qui peut me aider? bisou Rita

nouvelles?

Alors moi aussi je veut dire la mienne sur l'object. Je pense que donner seulement des croquettes soit pas bon pour les chiens, et c'est une chose bien connu que la majoritè des croquettes en commerce c'est "pas terrible" Moi j'ai alimentè Anthos pour 14 ans a nourriture maison et franchement j'avait une chienne tres contente!!! elle supportè tres male on plus les croquettes meme les meilleurs et plus cheres le resultat c'a ete toujour le meme: dhiarree! Pour mon nouveau petit Fanfan je fait comme So croquettes (une linea natural holistic) mais aussi viande poisson legume et fruit fresh.

Cherche de rester cool tout ira bien

Je voudrais partager avec vous cette article tres interessant que j'ai reçu par email Thu, April 29, 2010 3:27:49 AM[Berner-l] Osteosarcoma --------- - Bone Cancer in Dogs - What is the State of Practice and New Research? Jaime F. Modiano, VMD, PhD Director, Animal Cancer Care and Research Program, University of Minnesota In this article, I will briefly review what is bone cancer, how it is diagnosed, options for treatment, expected outcomes, and new information that will help us improve strategies for prevention, control, and treatment of primary osteosarcoma in dogs and children alike. Introduction Bone cancer can occur as a primary disease (originates from cells that normally reside in the bone space) or as a metastatic disease (spreads from cancers that arise elsewhere). In humans, most bone malignancies are metastases that arise from tumors outside the bone (breast, prostate). Primary bone tumors are less common, and osteosarcoma (primary bone cancer arising from bone-forming cells) is an orphan disease, meaning that these diseases have such a low prevalence that a general practice physician would not be expected to see more than one case in a year. In dogs, bone cancer also can occur as a primary or metastatic disease, but in contrast to humans, the most common form of bone cancer seen in dogs in the U.S. is osteosarcoma. This is probably due to various factors, including a higher relative risk in large and giant breed dogs to develop the disease over their lifetime (as compared to other dogs, and also to humans), as well as to the low incidence of mammary cancer in female dogs in the U.S. due to the practice of spaying and the relatively low incidence of other carcinomas in dogs that spread to bone in general, such as prostate, lung, colon, and renal cancer as well as of other common cancers of bone such as multiple myeloma. The remainder of this review will focus on the biology and treatment of osteosarcoma. Osteosarcoma occurs in humans, dogs, and cats. In people, it is predominantly a pediatric disease with peak onset by ~15 years of age. It is infrequent in adults, and incidence increases somewhat with age, with a second smaller peak after age 60. Primary osteosarcoma is a rare tumor, with fewer than 1,000 diagnoses per year. However, because of the demographics of the disease (i.e., the adolescent peak), it is considered an oncologic priority. Osteosarcoma is much more common in dogs than in people (~15 times). An estimated ~10,000 new diagnoses are made yearly, mostly in large and giant breed dogs, and it is seen only rarely in cats. Osteosarcoma accounts for approximately 85% of bone tumors in dogs. The median age at diagnosis is ~8 years, with a small peak of incidence in young animals (younger than 3 years). Still when the effect of body mass is taken into account, the overall risk for any dog to develop primary osteosarcoma is not magnified with increasing age. Dogs heavier than 90-lb account for almost 1/3 of cases, and most tumors in this group occur in the appendicular skeleton (limbs). Dogs under 30-lb account for less than 5% of cases, and in this group, most osteosarcoma occurs in the axial skeleton. In cats, there is no association with size or breed and the frequency of axial tumors is about the same as skeletal tumors. In dogs, appendicular osteosarcoma occurs in the metaphysis (at the site of the growth plates), "near the knee and away from the elbow." Occasionally, osteosarcoma will occur in the digits. Axial osteosarcoma can occur in any bone outside the limbs (skull, ribs, spine). Extraskeletal tumors are rare; in humans they occur almost exclusively in adults and most often in the skin. In dogs they can arise anywhere, including visceral organs (liver, spleen, heart), eyes, etc. Etiology and Risk Factors of Osteosarcoma A major component of this disease in dogs, and possibly in people, appears to be genetic (i.e., heritable). Risk is most accurately defined by body mass, although there is a direct correlation with size as well. In children, osteosarcoma is frequently seen in kindreds with mutations of the retinoblastoma susceptibility gene (RB-1), and this risk is paternally imprinted. In dogs, there are clear breed predispositions. A recent study by Phillips and colleagues published in Genomics (Phillips et al., 2007) showed that the narrow heritability in Scottish Deerhounds was 0.69; in other words, almost 70% of the cause is due to heritable traits. Narrow heritability (h2) is the proportion of the total variability due to genetic factors. It is not surprising heritable factors account for a significant component of risk in Scottish Deerhounds; more than 15% of dogs from this breed die from osteosarcoma. The best-fit model for inheritance of the risk traits in Scottish Deerhounds was a Mendelian major gene with dominant expression. Furthermore, Comstock and colleagues (Comstock et al., 2006) reported at the 2006 Genes Dogs and Cancer meeting (Chicago, IL) there are 4 regions of the genome that appear to be associated with an increased risk of osteosarcoma in Rottweilers, another breed where risk appears greater than what would be attributable to size alone (incidence estimated at more than 12%). Environmental factors that increase risk for osteosarcoma include rapid growth (therefore "large breed" puppy food has reduced levels of available energy to increase the time needed for these dogs to achieve their full size and mass potential), gender (the risk for males is 20 - 50% greater), and metallic implants to fix fractures. Chronic trauma and microscopic fractures have been proposed as risk factors, but this is difficult to prove conclusively. There was a study from David Waters group (Cooley et al., 2002), where survey data provided by owners showed an increase in risk to develop osteosarcoma in dogs that were spayed or castrated at an early age. The relative risk estimated from this study was as high as 4-fold higher for dogs neutered before one year of age than for intact dogs. Glickman's group published similar data in 1998 based on analysis of cases in the Veterinary Medical Database (Ru et al., 1998). These studies generated significant debate and concern among veterinarians and owners. Nevertheless, the results have not been reproduced consistently in other large population studies (for example, Phillips et al and Scottish Deerhounds). While these results may have increased some owners' reluctance to neuter or spay dogs, the possible 3-fold increase in risk of osteosarcoma in females should be placed in context of the 80 - 260-fold reduced risk of mammary cancer by early spaying, and the possible 4-fold increase in risk in males should be placed in context of behavioral problems, such as territorial aggression, roaming, marking behavior, and physiological problems such as prostatic hyperplasia and testicular cancers that appear more commonly (or exclusively) in intact male dogs. Natural History of Canine Osteosarcoma There are three common histologic types of osteosarcoma: osteoblastic, where tumor cells produce large amounts of tumor osteoid; chondroblastic, where tumor cells produce cartilage (chondroid) in addition to some amount of tumor osteoid (without osteoid the diagnosis is chondrosarcoma) ; and fibroblastic, where tumor cells are predominantly fibroblasts and can produce both collagen and tumor osteoid. The disease is highly metastatic, with distant spread mostly to lungs and other sites in bone. Osteosarcoma can also metastasize to lymph nodes and intra-abdominal organs. The metastatic pattern is similar for dogs and humans. Diagnosis of Canine Osteosarcoma Diagnosis is based on clinical signs, imaging, and biopsy. The clinical signs for appendicular osteosarcoma range from mild lameness with some evidence of pain to pathological fractures. The signs for axial and extraskeletal osteosarcoma are site-dependent. Imaging includes survey radiographs, and may be supplemented by magnetic resonance imaging (MRI) and/or computed tomography (CT) and nuclear scintigraphy. Imaging studies should include the primary tumor site and common sites of metastasis. Radiographic signs of osteosarcoma can range from severe lysis to severely sclerotic (increased density or hardening) lesions with new bone formation. There is usually loss of trabecular (internal) detail and indistinct demarcation of the tumor, associated soft tissue swelling, lysis of the outer boundary (cortex), and exuberant periosteal reactions that form the so-called "Codman's triangle." Although this is seen commonly, it is not always present and should not be considered the major determinant to make or rule out a diagnosis. Osteosarcoma rarely crosses joint space, except for an unusual type of necrotizing osteosarcoma of the tibia that is seen in Scottish Terriers and other smaller dogs. Nuclear scintigraphy is very sensitive, but not specific to identify lesions associated with osteosarcoma, as any region of osteoblastic (bone growth or remodeling) activity will be identified (i.e., arthritis). Nuclear scintigraphy is useful to determine the extent of primary tumor involvement. Fine needle aspiration cytology is commonly used as an adjunct to confirm a radiographic diagnosis. Cytology alone is generally not sufficient to make a definitive diagnosis, but the presence of "flag cells" with eosinophilic material, granular cells, and variable cell size and shape can support the diagnosis. Definitive diagnosis requires a biopsy, which can be obtained through an open incisional biopsy, a trephine biopsy, or a Jamshidi bone marrow biopsy needle. The diagnostic accuracy is almost 100% for open biopsies, ~95% for trephines, and >90% for Jamshidi needle biopsies. Biopsies should be obtained from center of lesion, and if a limb-sparing procedure is elected, the surgeon performing the surgery should perform the biopsy whenever possible. The pathologist will define the cell type (osteoblastic, chondroblastic, fibroblastic, mixed), grade (pleomorphism, proliferative fraction, etc.), and verify the presence of tumor osteoid, which is diagnostic. Other confirmatory tests can include immunohistochemistr y, staining for osteocalcin, osteonectin, and alkaline phosphatase (ALP). Staging and Prognosis for Canine Osteosarcoma Staging uses the "TNMG" (tumor, node, metastasis, grade) system. Stage I includes low-grade tumors (G1) without evidence of metastasis; stage II includes high-grade tumors (G2) without metastasis; and stage III includes dogs with metastatic disease. Substages "a" and "b" reflect intramedullary lesions (T1) and local extramedullary spread (T2), respectively. Most dogs with osteosarcoma are diagnosed in Stage IIb. In children, the site of primary disease is prognostic with tumors in the distal extremity carrying the best prognosis, tumors in the distal femur carrying intermediate prognosis, and tumors in the axial skeleton carrying the worst prognosis. In dogs, tumors of the mandible and scapula carry the best prognosis with a median survival of ~18 months, appendicular tumors have intermediate prognosis with a median survival of ~11 months, tumors of the spine and skull carry a worse prognosis with a median survival of ~6 months, and extraskeletal tumors carry the worst prognosis with a median survival of ~2 months. Tumor size is prognostic (the larger the tumor, the worst the prognosis), as is age (younger dogs do worse). Serum ALP levels also are predictive. Dogs with pre-operative levels of ALP> 110 U/L carry a worse prognosis than dogs with ALP

Oui So merci!

oui moi aussi je pense que il est pas fait pour les expos! du rest meme ma Anthos elle a etè trop expo!!! dans mon destin y'l a que des bouviers qui ils n'aiment pas montrer leur dents

Bonjour a vous tous on est rentrè de l'Italie l'expo de Valeggio a etè tres belle on plus on a eu droit a un superb soleil!!! Apres la disastrose experience de Fanfan a la national d'elevage je l'ai remis en ring pour eviter que le mauvaise experience du ring ça lui reste dans la tete Cette fois mme le judge a eu plus de patiente et on est arrivè a avoir un judgement! Le resultat c'etè "BON" il sareit mieux si il avait ouvert la bouche tout suite!! ahahah moi, je suis contente donc!!! ici quelque photos

gros bisou a toi et ta famille courage, perde pas l'espoir